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121 Possible Causes for Absent Ankle Reflex, Cerebellar Gait Ataxia

  • Peripheral Neuropathy

    All patients have gait ataxia and the majority have lower limb ataxia.[] Most people with PN have reduced or absent ankle reflexes (involuntary responses to a stimulus).[] Classically, ankle jerk reflex is absent in peripheral neuropathy.[]

  • Chronic Alcoholism

    The cause of the alcoholic gait is brain damage called alcoholic cerebellar ataxia.[] Both balance and gait are compromised. Can an Alcoholic Recover from Cerebellar Ataxia?[] However, long-term alcoholics frequently develop cerebellar ataxia. It’s called cerebellar ataxia, because it affects a part of the brain called the cerebellum.[]

  • Friedreich Ataxia

    Rehabilitation Of Limb And Gait Ataxia In Hereditary Cerebellar Ataxias: A Pilot Open-Labeled Study ,” published in the journal Neurological Sciences.[] Tendon reflexes are absent in almost all cases but may be weakly present if the patient is examined early in the course of the disease.[] […] in patients affected by hereditary cerebellar ataxias.[]

  • Charcot-Marie-Tooth Disease

    […] and kinetic cerebellar ataxia, nystagmus and dysarthria, she being wheelchair bound.[] Five months after the transient CNS symptoms, he finally developed signs and symptoms of neuropathy in the form of absent ankle reflexes.[] Ankle reflexes were bilaterally absent. Sequencing revealed a novel heterozygous c.712C T (p.R238C) mutation in the GJB1 gene.[]

  • Cerebellar Ataxia

    A 45-year-old man with a cerebellar gait ataxia, dysmetria, nystagmus and mild cerebellar dysarthria was diagnosed with insulin-dependent diabetes mellitus a year after the[] The association of brisk jerks and absent ankle reflexes may occur.[] Important neurologic signs other than cerebellar ataxia include peripheral neuropathy (decreased or absent tendon reflexes and decreased ankle reflexes); movement disorders[]

  • Hereditary Areflexic Dystasia

    […] neurologic deterioration Confusion Rigidity Hypertonia Spasticity Gait instability, worse in the dark Distal sensory impairment of all modalities Distal sensory loss of all[] Reflexes are normal in approximately two thirds of patients. In one study, ankle jerks were absent in 37.5%, and areflexia occurred in 12.5%.[] […] atrophy Cerebral amyloid angiopathy Abnormality of the adrenal glands Senile plaques Neurofibrillary tangles Alzheimer disease Truncal ataxia Sensorimotor neuropathy Progressive[]

  • Alcoholic Neuropathy

    Some people may experience frequent falls and gait unsteadiness due to ataxia.[] Limb or gait ataxia was prominent in 3 patients. Ataxia was of sensory type, and 1 patient additionally showed features of cerebellar ataxia.[] This ataxia may be caused by cerebellar degeneration, sensory ataxia, or distal muscle weakness.[]

  • Arsenic-Induced Polyneuropathy

    Some people can experience frequent falls and gait unsteadiness due to ataxia caused by cerebellar degeneration, sensory ataxia, or distal muscle weakness.[] Proximal lower limb muscles were affected in 40% patients, rare upper limb involvement and ankle reflexes were absent in all with evidence of axonal degeneration and reinnervation[] .  Tendon reflexes: Absent or Preserved except at ankles  Recovery: With delay after removal of toxin  Progression ("coasting") for 1 to 4 months  Improvement: Months[]

  • Autosomal Recessive Spastic Ataxia Type 2

    Main inclusion criteria for HA were cerebellar gait and/or limb ataxia, and for HSP, spasticity in the lower limbs, brisk reflexes and positive Babinski sign [ 11, 12 ].[] ankle jerks after 25 years of age Early non-progressive signs Increased deep tendon reflexes Bilateral abnormal plantar response Marked saccadic alteration of ocular pursuit[] Abstract Background: Hereditary cerebellar ataxias (HCA) and hereditary spastic paraplegias (HSP) are two groups of neurodegenerative disorders that usually present with progressive[]

  • Tabes Dorsalis

    In its severe form, this gait can cause an ataxia that resembles the cerebellar ataxic gait.[] The patellar and ankle reflexes were absent and there was loss of vibratory and position sense in the lower extremity.[] Gait was ataxic and of the stamping type. There were no cerebellar signs.[]

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