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31 Possible Causes for Acute Respiratory Failure, Compound Muscle Action Potential with Low Amplitude

  • Lambert Eaton Myasthenic Syndrome

    The patient presented with acute respiratory failure, making identification and management of her illness challenging.[] Distinguishing features of the RNS test in LEMS and MG are confirmed in this direct comparison study: the low compound muscle action potential amplitude, decrement at LRS,[] However, rare cases of severe respiratory compromise or respiratory failure have been reported in patients with LEMS.[]

  • Tick Paralysis

    It typically presents as an acute ascending paralysis occurring a few days after tick attachment and may result in respiratory failure and death.[] Neurophysiological studies reveal low-amplitude compound muscle action potentials with normal motor conduction velocities, normal sensory studies and normal response to repetitive[] failure and death.[]

  • Infantile Botulism

    Abstract Infantile botulism is a recently recognized cause of acute hypotonic paresis and respiratory failure in young infants.[] We conclude that the findings of low compound muscle action potential amplitude in combination with tetanic facilitation or posttetanic facilitation and absence of posttetanic[] […] tetraparesis and respiratory failure hypotonia head lag ptosis reduced facial expression reduced suck and swallow loss of reflexes slowly improves after weeks Adult intestinal[]

  • Guillain-Barré Syndrome

    CASE REPORT: An 82-year-old woman presented with acute ascending flaccid paralysis and acute respiratory failure.[] muscle action potentials reduced amplitude with relatively preserved conduction velocity implicates axonal neuropathy delayed/absent F waves implicates nerve root involvement[] Approximately 30% of patients require intubation and ventilation because of respiratory failure.[]

  • Myasthenia Gravis

    respiratory failure after a 12-day hospitalization.[] Distinguishing features of the RNS test in LEMS and MG are confirmed in this direct comparison study: the low compound muscle action potential amplitude, decrement at LRS,[] A decremental pattern ( 10% difference in compound muscle action potential amplitude between the first and fourth or fifth stimulus) is the usual finding.[]

  • Dermatomyositis

    ECMO was giving when the patient suffered acute respiratory failure.[] Low compound muscle action potentials (CMAP) amplitudes may not necessarily indicate a neuropathic process, as it may reflect muscle atrophy and fibrosis.[] METHODS: We retrospectively enrolled 14 patients with CADM-associated RPIPs and acute respiratory failure treated with PMX-DHP, corticosteroids, and immunosuppressive agents[]

  • Thyrotoxic Periodic Paralysis

    TPP should be kept in mind as a cause of acute respiratory failure in association with acute muscle weakness to avoid delayed diagnosis and improper management.[] […] electrical compound muscle action potential on electromyography and no notable changes in amplitudes after low doses of epinephrinine; and typical acid-base and electrolyte[] Arterial blood gas indicated acute hypercapnic respiratory failure (pCO(2): 118.9 mmHg), and ventricular tachycardia was documented by electrocardiogram.[]

  • Neuropathy

    Goldenberg and Steven White, Neuromuscular Disorders and Acute Respiratory Failure: Diagnosis and Management, Neurologic Clinics, 30, 1, (161), (2012).[] Patients have relatively normal nerve conduction velocities but low amplitude sensory nerve action potentials and compound muscle action potentials.[]

  • Food-Borne Botulism

    Rapid progression to respiratory failure and paralysis is seen in serotype F. [ 5 ] Signs Acute onset of bilateral cranial nerve involvement. Failure of accommodation.[] Electrophysiologic tests of such patients show reduced compound muscle action potentials (CMAPs), low amplitudes and short durations of motor unit potentials (MUPs), and mild[]

  • Bickerstaff Brainstem Encephalitis

    One patient succumbed to respiratory failure allowing pathological studies to be done.[] (absent R2) suggested central dysfunction, whereas results of facial nerve conduction studies (low amplitudes of compound muscle action potentials), F-wave and H-reflex studies[] This showed evidence of severe myelin destruction with perivascular mononuclear infiltration and a normal brain stem, features in keeping with an acute infective polyneuritis[]

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