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89 Possible Causes for Astrocytic Gliosis, Psychiatric Manifestation

  • Alzheimer Disease

    Dementias with prominent psychiatric-behavioral manifestations and gait impairment have a faster progression compared to AD.[] Multifocal spongiform encephalopathic changes, mononuclear perivascular infiltrates, subcortical demyelination and gliosis were also found.[] Behavioral and psychiatric manifestations in dementia patients in a community: caregiver burden and outcome.[]

  • Chronic Fatigue Syndrome

    Manifestations that were commonly reported included neurological manifestations (84.2 %), musculoskeletal (78.9 %), psychiatric (63.1 %), fatigue (63.1 %), gastrointestinal[] PubMed Google Scholar Hwang SY, Jung JS, Kim TH, Lim SJ, Oh ES, Kim JY, Ji KA, Joe EH, Cho KH, Han IO: Ionizing radiation induces astrocyte gliosis through microglia activation[] In a typical severe case the early manifestations included localized muscular weakness, stiffness of the neck and back, headache, diarrhea and temperature elevation.[]

  • Primary Progressive Multiple Sclerosis

    As lesions evolve, there is prominent astrocytic proliferation (gliosis) [ 11 ].[]

  • McLeod Neuroacanthocytosis Syndrome

    manifestations, and distinct striatal imaging findings.[] Marked neuronal loss and astrocytic gliosis were observed on histologic examination.[] A cell loss of 90% in the striatum with astrocytic gliosis has been reported.[]

  • Hypoxia

    After 20 weeks' gestation, hypoxic-ischemic insults result in astrocyte activation with subsequent gliosis.[] GFAP is a useful marker to study astrocytic response to injury.[] The 3 major features of status marmoratus include neuronal loss, gliosis and hypermyelination.[]

  • Multiple Sclerosis

    Psychiatric manifestations of multiple sclerosis: A review. Can J Psychiatry 1996 ; 41: 441 – 445. Google Scholar SAGE Journals ISI 5. Patten, SB, Svenson, LW, Metz, LM.[] Multiple sclerosis (MS) is characterised by inflammation, demyelination, axonal or neuronal loss, and astrocytic gliosis in the central nervous system (CNS), which can result[] Fatigue and psychiatric symptoms, such as depression or euphoria, can also be manifestations of MS. Symptoms (attacks) of MS need disseminated in “time and space.”[]

  • Huntington's Disease

    Huntington's disease (HD) is an autosomal dominant and progressive neurodegenerative syndrome characterized by motor, cognitive and psychiatric manifestations.[] Proliferation of reactive astrocytes causes the affected nuclei to appear more cellular than normal.[] Although noncompliance to oral antipsychotic drugs can be a practical problem, especially when significant psychiatric manifestations of HD are present, the effect of depot[]

  • Boxer's Dementia

    Pathophysiology Although the details remain to be elucidated, there is mounting evidence that reactive gliosis and redistribution of aquaporin 4 molecules, leading to disruption[] There is severe neuronal loss and gliosis (A) with neurofibrillary tangles (NFTs) (white arrow, B ) in the SN.[] […] traumatic encephalopathy is characterized by the presence of neurofibrillary tangles and TDP-43 binding protein in subcortical and perivascular regions, often with reactive astrocytes[]

  • Subacute Sclerosing Panencephalitis

    Psychiatric manifestation of SSPE. J Neuropsychiatry Clin Neurosci 2006;18:560. [ PUBMED ] 3. Kartal A, Kurt AN, Gürkas E, Aydin K, Serdaroglu A.[] There is often evidence of neuronal degeneration, gliosis, proliferation of astrocytes, perivascular cuffing, lymphocytic and plasma cell infiltration, and demyelination.[] In the earlier stages of the disease, psychiatric manifestations, such as behavioral changes, mood swings, depression, and cognitive decline, are noted.[]

  • Anti-NMDA Receptor Encephalitis

    OBJECTIVE: Anti-N-methyl-d-aspartate receptor (NMDA-R) encephalitis is a recently discovered disorder with prominent psychiatric manifestations that is often misdiagnosed.[] […] of various serum proteins and the reactivity of proximal microglia and astrocytes in the uninjured brainstem and spinal cord enlargements after 1-4 weeks.[] Acute neurological and psychiatric manifestations are the common presenting symptoms.[]

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