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32 Possible Causes for Behavior Problem, Bilateral Ankle Clonus, Cerebellar Gait Ataxia

  • Primary Progressive Multiple Sclerosis

    […] and visual defects, muscular weakness, absent abdominal reflexes, hyperactive tendon reflexes, cerebellar ataxia, retrobulbar neuritis, loss of proprioceptive sense, spastic[] […] due to severe intention tremor Hypotonia: Loss of muscle tone Gait ataxia: Loss of balance Dysmetria : Inability to coordinate movement in which your either overshoot or[] […] tendon reflexes; cerebellar ataxia; retrobulbar neuritis; loss of proprioceptive sense; spastic weakness of legs; vertigo; the “classic” Charcot’s triad (dysarthria, nystagmus[]

  • Huntington's Disease

    […] dysfunction masquerading dominant spinocerebellar ataxias (SCA), were occasionally reported.[] Behavioral Problems in Huntington’s Disease. A helpful guide for coping with and caring for individuals experiencing the behavioral symptoms of HD. Paulsen, J.[] Unlike cerebellar ataxias, determining the precise age at onset of disease in Huntington disease ( Penney et al ., 1990 ) may be difficult due to the overlap of psychiatric[]

  • Subacute Combined Degeneration of Spinal Cord

    It causes ataxia of stance and gait with relative sparing of the arms. It has an insidious onset and a subacute or chronic course.[] Midline cerebellar degeneration (also referred to as alcoholic cerebellar degeneration) is a component of WKS, but may also occur alone.[]

  • Cerebral Palsy

    GA usually presents with pure cerebellar ataxia primarily affecting gait and lower limbs.[] Some behavioral problems are extreme irritability, feeding difficulties, persistent gagging or choking when fed after 6 months of age especially if the tongue pushes the food[] Children with CP may have additional problems, including the following: Seizures Vision, hearing, or speech problems Learning disabilities and behavior problems Mental retardation[]

  • Cerebral Palsy with Spastic Diplegia

    ., hyperactive deep tendon reflexes, bilateral ankle clonus, extensor toe signs)[see above].[] Ataxia is associated with cerebellar lesions. Ataxia is often combined with spastic diplegia. Most ataxic children can walk but some need walkers.[] Children with CP may also have additional problems, including: seizures vision loss or impairment hearing loss or impairment speech impairment learning disabilities behavioral[]

  • Spastic Paraplegia

    The deep tendon reflexes were increased in all four limbs, but Babinski sign was not elicited bilaterally. Neither ankle nor knee clonus was observed.[] Main inclusion criteria for HA were cerebellar gait and/or limb ataxia, and for HSP, spasticity in the lower limbs, brisk reflexes and positive Babinski sign [ 11, 12 ].[] Another five patients had behavioral problems in childhood. Brain MRIs from nine patients were available for review.[]

  • Creutzfeldt Jakob Disease

    ataxia, gait abnormalities, dementia, dysarthria, ocular dysmetria, and hyporeflexia or areflexia in the lower extremities.[] Memory problems, behavior changes, vision problems, and poor muscle coordination progress quickly to dementia , coma, and death. Most patients die within a year.[] A 62-year-old man presented with abnormal behavior and cognitive impairment.[]

  • Dementia

    Hoffman sign and ankle clonus.[] All patients have gait ataxia and the majority have lower limb ataxia.[] Behavioral problems are seen in most patients with dementia and are often poorly characterized in the literature.[]

  • Progressive Multifocal Leukoencephalopathy

    The patient was diagnosed as having bilateral ankle clonus.[] , including ataxia and dysmetria JC virus encephalopathy presents with global cerebrocortical dysfunction accompanied by seizures ( Cleve Clin J Med 2011;78:S8 ) Diagnosis[] Behavioral health problems including depression, suicidal thoughts or hallucinations.[]

  • Progressive Supranuclear Palsy

    Reflexes were hyperreflexic but symmetric in the upper extremities with unsustained ankle clonus. Extensor toe signs were absent.[] A Japanese male patient presented with gait disturbance at the age of 69 years. His principal symptom was cerebellar ataxia for several years.[] The patient presented with loss of recent memory, abnormal behavior and change in personality at the age of 60. The symptoms were progressive.[]

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