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129 Possible Causes for Bilateral Babinski's Reflex, Hyperreflexia

  • Upper Motor Neuron Disease

    Upper Motor Neuron Disease Symptoms The weakness of involved muscles, hyperreflexia, hypertonicity, mild disuse atrophy, and abnormal reflexes; damaged tracts are in the lateral[] , Babinski Decreased or absent: hyporeflexia, cutaneous reflexes decreased or absent Involuntary movements Muscle spasms: flexor or extensor With denervation: fasciculations[] No spasticity or hyperreflexia.[]

  • Spastic Paraplegia

    On neurological examination, he showed hyperreflexia, spasticity, and weakness in the lower extremities and bilateral Babinski reflexes.[] Hyperreflexia, extensor plantar response and increased muscle tone are the recognized UMN signs where as the weakness, muscle wasting, hyporeflexia, muscle cramps, and fasciculations[] People with this condition can also experience exaggerated reflexes (hyperreflexia), ankle spasms, high-arched feet (pes cavus), and reduced bladder control.[]

  • Pyramidal Tract Lesion

    Symptoms include an increase in the muscle tone in the lower extremities, hyperreflexia, positive Babinski and a decrease in fine motor coordination.[] These include hyperreflexia, spasticity, positive Babinski sign and loss of superficial abdominal reflexes.[] […] decussation causes impairment of movement in the opposite body-half, which is especially severe in the arm and leg and is characterized by muscular weakness, spasticity and hyperreflexia[]

  • Spinal Cord Compression

    Physical examination revealed hyperreflexia and a T11 sensory level. MRI revealed a pseudomeningocele compressing the thoracic spinal cord.[] Late signs include demonstrable weakness, clear sensory loss, bilateral Babinski signs, and decreased anal sphincter tone and bulbocavernosus reflex.[] Lhermitte's sign (intermittent shooting electrical sensation) and hyperreflexia may be present.[]

  • Spinal Cord Infarction

    Hyperreflexia may follow initial symptoms, and a positive Babinski reflex may be elicited.[] Neurologic examination demonstrated a broad-based, spastic gait, brisk muscle stretch reflexes, and bilateral Babinski signs.[] Initially areflexia is present due to spinal shock but, hyperreflexia and spasticity appear later The most common form is anterior spinal artery syndrome.[]

  • Spinal Cord Neoplasm

    As spinal cord compromise advances, hyperreflexia and Babinski reflexes are typically present. Lax rectal sphincter tone is a late sign of spinal cord dysfunction.[] Compression, motor deficits are more common than sensory Pain on Vertebra l body compression Early changes Flaccidity Hyporeflexia Later changes Cauda Equina Syndrome Spasticity Hyperreflexia[] Late signs include demonstrable weakness, clear sensory loss, bilateral Babinski signs, and decreased anal sphincter tone and bulbocavernosus reflex.[]

  • Hepatic Encephalopathy

    […] cause mitochondrial dysfunction Clinical features Behavioral and personality changes, including irritability and disinhibition Motor abnormalities including hypertonia, hyperreflexia[] […] posturing; muscle spasticity; and bilateral extensor plantar reflexes (see reflex, babinski).[] […] of earliest clinical symptoms is disturbance in diurnal sleep patterns e) Spectrum of presentation is broad, but some signs to be aware of are: bradykinesia, asterixis, hyperreflexia[]

  • Pallidopyramidal Syndrome

    Clinical manifestations include tremor, bradykinesia, rigidity, postural instability, spasticity, mainly in the lower limbs, and hyperreflexia.[] , bilateral Babinski reflex Good Blood tests, comprehensive of calcium and phosphate, were normal.[] The pyramidal features include paraparesis, scissoring of gait, Bakinski sign, hyperreflexia, and pseudobulbar effect.[]

  • Myelitis

    Examination reveals moderate paraparesis with hyperreflexia, a left extensor plantar response, impaired vibratory and proprioceptive sensation, and a sensory level at T6.[] Reflexes were diminished bilaterally and Babinski sign was equivocal. Examination of optic fundi and cranial nerves were normal.[] Sx: leg weakness, sphincter dysfunction, abnormal DTRs [acutely may be absent, but hyperreflexia the rule].[]

  • Friedreich Ataxia

    We report two patients with delayed-onset, hyperreflexia and gradually progressive disease.[] His reflexes were absent and Babinski sign was present bilaterally. A nine-year-old sister was found to have mild ataxia but was otherwise neurologically intact.[] His examination was significant for spastic gait, hyperreflexia, and sensory neuropathy. Genetic testing revealed a compound heterozygous mutation in the FXN gene.[]

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