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7,020 Possible Causes for bone, development, marrow

  • Osteoporosis

    KEYWORDS: anorexia nervosa; bone marrow edema; femoral neck fracture; osteoporosis; pregnancy[] The WHO has developed an algorithm for estimation of 10-year fracture risk which may be used even in the absence of bone mineral density.[] Excerpt This first-ever Surgeon General’s Report on bone health and osteoporosis illustrates the large burden that bone disease places on our Nation and its citizens.[]

  • Epstein-Barr Virus-Associated Lymphoproliferative Disorder

    Their occurrence is rare after autologous bone marrow transplantation (BMT) with only five published reports in the literature.[] In this patient, vorinostat therapy also appears to be linked to the development of an EBV-related lymphoproliferative disorder.[] Bone marrow examination showed marked hypoplasia with 45.2% infiltration of CD3 , CD8 , CD16 and CD57 granular lymphocytes.[]

  • Sinusitis

    Infectious Diseases, Kaiser Permanente Medical Center, Los Angeles, CA, USA. 2 Department of Ophthalmology, Kaiser Permanente Medical Center, Los Angeles, CA, USA. 3 Bone Marrow[] There have been several recent developments in the understanding of mechanisms, diagnosis, and treatment of odontogenic sinusitis, and clinicians should be aware of these[] Anatomic pathology identified necrotic bone with invasive fungal hyphae.[]

  • Acute Myelocytic Leukemia

    Chromosome analysis of the direct bone-marrow preparation showed 100% of cells with trisomy 13 in the first and 10% of cells in the second.[] Abstract In our hospital within one year two patients with Crohn's disease were seen who developed an acute myelocytic leukemia.[] Abstract A 28-year-old man developed Crohn's disease and myelodysplastic syndrome concurrently. Chromosomal analysis of the bone marrow revealed a normal male karyotype.[]

  • Myelofibrosis

    Bone marrow aspiration is usually dry. A bone marrow biopsy is required to show fibrosis.[] Finally, the JAK2 inhibitors no longer in clinical development are summarized in tabular form.[] Several questionnaires have been developed for patient self-report of MF symptoms in clinical trials and each includes unique instructions, items, and/or response scales.[]

  • Fanconi Anemia

    Abstract More than 90% of Fanconi anemia (FA) patients experience progressive bone marrow failure during life with a median onset at 8 years of age.[] The patients have very high risks of development of malignancies ( Fig. 7 ).[] KEYWORDS: Fanconi anemia; bone marrow transplant; renal insufficiency; renal transplant[]

  • Acute graft versus host disease

    This is called an autologous bone marrow transplant. Bone marrow cells are a major component of the immune system.[] Multivariate analyses identified the absolute number of MAIT cells ( high (  0.48/μL) group developed aGVHD, five within the first 30 days post HSCT.[] Higher levels of β2-m were observed in patients who developed aGVHD (P .008).[]

  • Graft-versus-Host-Disease

    This is called an autologous bone marrow transplant. Bone marrow cells are a major component of the immune system.[] Development of cGVHD while on intensive immunosuppression for aGVHD indicates resistance by definition because it develops through treatment.[] GVHD is not always a negative development following a blood and bone marrow transplant.[]

  • Acute Leukemia

    We herein describe a case of rhabdomyosarcoma with bone marrow metastasis mimicking acute lymphoblastic leukemia.[] We report a case of a 45-year-old man who developed extramedullary acute leukemia with a myeloid immunophenotype (myeloid sarcoma) with its initial presentation within an[] […] and those HLA alleles that predispose to development of acute leukemia.[]

  • Aplastic Anemia

    Acquired aplastic anemia (AA) is a serious hematologic disorder characterized by pancytopenia and bone marrow aplasia or hypoplasia. 1 Bone marrow transplantation (BMT) and[] Developing a new QoL questionnaire specific for this patient group is warranted.[] BACKGROUND: Acquired aplastic anemia (AA) is characterized by deficiency or dysfunction of the bone marrow (BM) microenvironment.[]