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79 Possible Causes for Brandt Syndrome, Hypophosphatemia

  • Chronic Alcoholism

    Anemia Leukocytosis or leukopenia Thrombocytopenia Increased serum globulin levels Metabolic Elevated blood ammonia level Hyperglycemia Respiratory alkalosis Hypomagnesemia Hypophosphatemia[] Abnormality of a thiamine-requiring enzyme in patients with Wernicke-Korsakoff's syndrome. N. Engl. J. Med. 297: 1367–1370.[] PubMed Google Scholar Brandt, J., Butters, N., Ryan, C., and Bayog, R. (1983). Cognitive loss and recovery in chronic alcohol abusers. Arch. Gen. Psychiat. 40: 435–442.[]

  • Psychogenic Hyperventilation

    Tetany occurs because respiratory alkalosis causes both hypophosphatemia and hypocalcemia.[] Brandt T. Vertigo. Its multisensory syndromes. Springer Verlag, New York, 1991. Brandt T, Dieterich M, and Strupp M.[] Acute secondary hypophosphatemia is common and may contribute to paresthesias and generalized weakness.[]

  • Intravenous Hyperalimentation

    We have discussed pertinent literature related to neurologic dysfunction as a result of hypophosphatemia.[] The patients all developed hypophosphatemia (mean serum phosphate of 0.3 mg per 100, ml) and muscular weakness.[] A complication of the hypophosphatemia of parenteral hyperalimentation, N. Engl. J. Med. 290 : 1403. CrossRef Google Scholar Curreri, P.[]

  • Brandt Syndrome

    […] metabolism E83.2 Disorders of zinc metabolism E83.3 Disorders of phosphorus metabolism and phosphatases E83.30 Disorder of phosphorus metabolism, unspecified E83.31 Familial hypophosphatemia[] Brandt syndrome (BS) is also called acrodermatitis enteropathica (AE). It is a congenital disease that causes zinc deficiency due to malabsorption.[] Inclusion term(s): Acrodermatitis enteropathica E83.3 Disorders of phosphorus metabolism and phosphatases E83.30 Disorder of phosphorus metabolism, unspecified E83.31 Familial hypophosphatemia[]

  • Familial Hypophosphatemia

    Familial syndromes causing hypophosphatemia are considered to be rare in he general population.[] Celiac disease, cerebral calcifications and epilepsy syndrome. Arch Pediatr . 1996;3:1013–9. 28. Hagander B, Berg NO, Brandt L, Norden A, Sjolund K, Stenstam M.[] (Also known as/Synonyms) Familial Hypophosphatemic Bone Disease Phosphate Diabetes X-Linked Hypophosphatemia (XLH) What is Familial Hypophosphatemia?[]

  • Childhood Celiac Disease

    The FIP1L1-PDGFRA fusion gene cooperates with IL-5 to induce murine hypereosinophilic syndrome (HES)/chronic eosinophilic leukemia (CEL)-like disease.[] Yamada Y, Sanchez-Aguilera A, Brandt EB, McBride M, Al-Moamen NJ, Finkelman FD, Williams DA, Cancelas JA, Rothenberg ME Blood 112(6) 2500-2507 Sep 2008 [Refereed] Anti-IL-[] Yamada Y, Rothenberg ME, Lee AW, Akei HS, Brandt EB, Williams DA, Cancelas JA Blood 107(10) 4071-4079 May 2006 [Refereed] The alpha4bbeta7-integrin is dynamically expressed[]

  • Malignant Melanoma

    Westerdahl J, Olsson H, Masback A, Ingvar C, Jonsson N, Brandt L, et al. Use of sunbeds or sunlamps and malignant melanoma in southern Sweden.[] Risk of cutaneous malignant melanoma in patients with “classic” atypical-mole syndrome: a case-control study. Arch Dermatol . 1994;130:993–8. 17.[]

  • Iron Deficiency

    Analyzed safety concerns include gastrointestinal side effects (oral iron) and risks of hypotension, anaphylaxis, infection, hypophosphatemia, oxidative stress and mortality[] Brandt SJ, Bodine DM, Dunbar CE, Nienhuis AW. Dysregulated interleukin 6 expression produces a syndrome resembling Castleman’s disease in mice.[]

  • Infectious Colitis

    Professor Department of Pediatrics Director Yale Center for X-linked Hypophosphatemia Yale School of Medicine New Haven, Connecticut Jose R. Fernandez, Ph.D.[] Brandt LJ, Bjorkman D, Fennerty MB, et al. Systematic review on the management of irritable bowel syndrome in North America.[] Meta-analysis: the treatment of irritable bowel syndrome. Aliment Pharmacol Ther 2004; 20: 1253–69 PubMed CrossRef Google Scholar 10.[]

  • Anorexia Nervosa

    Herein, we describe a case of persistent recurrent hypophosphatemia in a male-to-female transgender patient with severe anorexia nervosa.[] A 4-year prospective study of Eating Disorder NOS compared with full eating disorder syndromes.[] Nutritional support was begun slowly, however electrolyte derangements began to manifest on hospital day 2, with hypophosphatemia, hypokalemia, hypocalcemia, and hypomagnesemia[]

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