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1,505 Possible Causes for Cerebellar Gait Ataxia, Nocturnal Leg Cramp, Uric Acid Increased

  • Chronic Alcoholism

    The cause of the alcoholic gait is brain damage called alcoholic cerebellar ataxia.[] Gout Gout is a type of arthritis caused by the formation of uric acid crystals in the joints. It can be very painful.[] Both balance and gait are compromised. Can an Alcoholic Recover from Cerebellar Ataxia?[]

  • Pernicious Anemia

    It causes ataxia of stance and gait with relative sparing of the arms. It has an insidious onset and a subacute or chronic course.[] Midline cerebellar degeneration (also referred to as alcoholic cerebellar degeneration) is a component of WKS, but may also occur alone.[]

  • Chronic Kidney Insufficiency

    Chronic kidney insufficiency affects a significant number of individuals in the developed countries, particularly the United States. The progressive nature of the disease and a substantial reduction in the quality of life over the period of time are the primary reasons why an early diagnosis is crucial. Anemia,[…][]

    Missing: Cerebellar Gait Ataxia
  • Restless Legs Syndrome

    All patients have gait ataxia and the majority have lower limb ataxia.[] Two common causes of pain or discomfort in legs are nocturnal leg cramps (NLC) and restless leg syndrome (RLS).[] Leg pain and discomfort are common complaints in any primary physician's clinic.[]

    Missing: Uric Acid Increased
  • Hyponatremia

    […] exhibit increased serum uric acid levels (greater than 0.3 mmol/L). 25 In contrast, in patients with SIADH, serum uric acid levels are actually depressed (less than 0.24[] […] syndrome of inappropriate antidiuretic hormone secretion (SIADH) (low to normal serum creatinine and urea, decreased uric acid levels, increased urine Na ). [35] However,[] RESULTS: Serum levels of sodium, chloride, phosphorous, uric acid, and osmolality were low. Serum α-fetoprotein, β-HCG, and lactate dehydrogenase were highly elevated.[]

    Missing: Cerebellar Gait Ataxia
  • Peripheral Neuropathy

    All patients have gait ataxia and the majority have lower limb ataxia.[] GA is usually of insidious onset; however, it can also be rapidly progressive mimicking paraneoplastic cerebellar degeneration. Gaze-evoked nystagmus is common.[]

    Missing: Uric Acid Increased
  • Electrolytes Abnormal

    A few years ago, researchers in the United Kingdom found that 300 mg of supplemental magnesium reduced nighttime or nocturnal leg cramps in individuals who suffered chronic[] Renal Failure: Electrolytes Elevated Electrolytes: potassium, phosphate, and magnesium Decreased Electrolytes: sodium, calcium Other Increases: urea, creatinine, uric acid[] Urea, creatinine, uric acid, sulfate, phosphate, phosphorus, lipids, cholesterol, neutral fats, and some amino/organic acids may accumulate, while albumin levels fall.[]

    Missing: Cerebellar Gait Ataxia
  • Amyotrophic Lateral Sclerosis

    FDA bans quinine for nocturnal leg cramps. Drug Utilization Rev. 1995; Oct:150. 26. Moss AH, Casey P, Stocking CB, Roos RP, Brooks BR, Siegler M.[] This study measured serum uric acid levels in a large number of ALS clinical trial participants, and by controlling patient body mass index, showed that higher baseline levels[] Stretching before sleep reduces the frequency and severity of nocturnal leg cramps in older adults: a randomised trial.[]

    Missing: Cerebellar Gait Ataxia
  • Hypokalemia

    acid, and amino acids.[] Much of this correlation is attributable to common causes (ie, diuretics, diarrhea), but hypomagnesemia itself may also result in increased renal potassium losses.[] […] acquired renal tubular diseases, such as the renal tubular acidoses and Fanconi syndrome, an unusual syndrome resulting in renal wasting of potassium, glucose, phosphate, uric[]

    Missing: Cerebellar Gait Ataxia
  • Pes Planus

    It presents with leg oedema, pain, nocturnal cramping, venous claudication, skin pigmentation, dermatitis and ulceratiaion (usually on the medial aspect of the lower leg).[] Gout is a form of arthritis caused by excess uric acid in the blood that most commonly affects the big toe joint. Symptoms Joint swelling, warmth, and redness.[] Increased pain and swelling in the morning, or after sitting or resting. Walking may be difficult. Treatment Pain relievers and anti-inflammatory medications.[]

    Missing: Cerebellar Gait Ataxia

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