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242 Possible Causes for Gait Apraxia, Prominent Sulci

  • Normal Pressure Hydrocephalus

    NPH is suggested when ventricular dilation is out of proportion to the prominence of the sulci, but this is a subjective call.[] INTRODUCTION: Normal pressure hydrocephalus (NPH), described by Hakim and Adams in 1965, is characterized by gait apraxia, urinary incontinence, and dementia.[] Normal pressure hydrocephalus (NPH) is characterized by gait apraxia, urinary incontinence, and dementia.[]

  • Creutzfeldt Jakob Disease

    B, Corresponding FLAIR image shows marked prominence of ventricles and sulci for the patient’s age.[] We report a 56-year-old right-handed man with rapidly progressive gait ataxia and involuntary elevation of the left upper limb.[] During the next few weeks, the patient developed cognitive impairment, apraxia, visual hallucinations, and myoclonus. He met diagnostic criteria for CJD.[]

  • Dialysis Dementia

    Abstract The brains of 7 patients treated with hemodialysis were studied. Four of these patients had the dialysis encephalopathy syndrome (DES). Senile plaques and or neurofibrillary tangles were found in 5 of the 7 cases, 3 with and 2 without DES. One case of each group had plaques to an extent compatible with[…][]

    Missing: Prominent Sulci
  • Communicating Hydrocephalus

    CT and MRI reveal bilateral extracerebral fluid collections, prominent sulci, normal ventricles, and no evidence of compression of the brain.[] The severity of the core clinical picture with predominant sensory ataxia, gait apraxia, lower limb spasticity, cognitive impairment and bladder dysfunction correlated with[] Pre-contrast axial CT Sulci and gyri are very prominent due to cortical atrophy, unlike in obstructive hydrocephalus where there is effacement of sulci and gyri.[]

  • Marchiafava Bignami Disease

    Findings CT shows mild prominence of the ventricles and sulci consistent with mild generalized cerebral volume loss.[] , aphasia, apraxia and incontinence with a resultant high morbidity and mortality rates.[] Other individuals may have acute, subacute, or chronic onset of dementia and/or gait problems.[]

  • Anterior Cerebral Artery Occlusion

    Paralysis or weakness of the contralateral foot and leg due to involvement of Motor leg area Cortical Sensory loss in the contralateral foot and leg Gait apraxia Impairment[] apraxia, incontinence, neglect, akinetic mutism and abulia, mood disturbance, aphasia, callosal disconnection syndromes, and pathological grasp phenomena and alien hand.[] Apraxia of gait is apraxia related to walking and may look like an unusually wide walk with short, flat steps.[]

    Missing: Prominent Sulci
  • Progressive Multifocal Leukoencephalopathy

    The image from day 258 shows prominence of cerebellar sulci (red arrow) and increased size of fourth ventricle.[] The differential diagnosis of PML should be considered in any immunocompromised patient presenting with impairment of consciousness, dysphagia, apraxia, gait disturbance,[] Examination Focal neurologic signs [Mostly related to occipital lobes] Aphasia Hemiparesis Ataxia Cortical blindness Limb apraxia Brainstem symptoms Head tremor Gait abnormalities[]

  • Aspartylglucosaminuria

    sulci Atrophy Normal 2 17.3 F Decreased Decreased Normal None Poor Atrophy Normal 3 26.6 F Decreased Decreased Mildly increased Several Poor Mild cerebral and cerebellar[] BACKGROUND: The pathogenesis of gait apraxia (GA) is unknown [10].[] Periventricular Juxtacortical GM/WM T2 SI T2 foci differentiation Cerebral and cerebellar atrophy CC atrophy PVS dilatation Other 1 12.0 F Decreased Decreased Increased None Poor Some prominent[]

  • Microangiopathy

    Furthermore, gait apraxia appeared predominantly in those with frontal lobe or basal ganglia lesions.[] Basal ganglia WMLs/lacunar infarcts related to more complaints of gait apraxia, vertigo and incontinence.[] Basal ganglia WMLs/lacunar infarcts were seen in patients with more complaints of gait apraxia, vertigo and incontinence.[]

    Missing: Prominent Sulci
  • Stroke

    Stroke Syndromes Symptoms and Signs Syndrome Contralateral hemiparesis (maximal in the leg), urinary incontinence, apathy, confusion, poor judgment, mutism, grasp reflex, gait[] apraxia Anterior cerebral artery (uncommon) Contralateral hemiparesis (worse in the arm and face than in the leg), dysarthria, hemianesthesia, contralateral homonymous hemianopia[]

    Missing: Prominent Sulci

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