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3,749 Possible Causes for Glucocorticoid

  • Polymyalgia Rheumatica

    Glucocorticoids are the primary therapy for PMR and GCA.[] RESULTS: All patients with PMR were deemed to respond to glucocorticoids clinically.[] Treatment with glucocorticoid (GC) is the preferred therapy for polymyalgia rheumatica (PMR), but some patients show poor responses to the initial GC regimen or experience[]

  • Cushing Syndrome

    ACTH up-regulation with paradoxical response to glucocorticoid, possibly through the hypomethylation of the POMC promoter, exacerbated the patient's condition.[] Glucocorticoid levels, serum parameters, and BMI were analyzed among a larger patient cohort including those with different forms of CS, APAs, and Cushing disease.[] Excess glucocorticoids can be from increased endogenous production or prolonged exposure to exogenous use of glucocorticoid products.[]

  • Adrenal Insufficiency

    glucocorticoids.[] KEYWORDS: adrenal insufficiency; dermatology; glucocorticoids[] Despite glucocorticoid replacement, patients with AI have reduced life expectancy and quality of life.[]

  • Addison's Disease

    Glucocorticoid receptor polymorphisms that alter glucocorticoid sensitivity may influence metabolic and cardiovascular risk factors in patients with AD.[] Supra-physiological doses of glucocorticoids and longer-standing disease (with a consequently higher glucocorticoid intake) might be the main causes behind patients' increased[] BACKGROUND: Uncertainty exists whether glucocorticoid receptor (GCR) polymorphisms play a role in steroid-related side effects in Addison's disease (AD) patients on hydrocortisone[]

  • Addisonian Crisis

    The diagnosis of acute hypoadrenalism seldom is considered in patients without known adrenal insufficiency who are taking supraphysiologic amounts of glucocorticoids.[] If you have been instructed by your doctor, be prepared to give yourself an emergency shot of glucocorticoid or to increase your dosage of oral glucocorticoid medicine in[] These enzymes degrade a wide range of metabolically active compounds including glucocorticoids.[]

  • Familial Hyperaldosteronism Type 1

    The disorder became known as glucocorticoid-remediable aldosteronism (GRA).[] Glucocorticoid remediable aldosteronism also describable as aldosterone synthase hyperactivity, is an autosomal dominant disorder in which the increase in aldosterone secretion[] ATUALIZAÇÃO Glucocorticoid-remediable aldosteronism Aldosteronismo remediável por glicocorticóide Graham T. McMahon; Robert G.[]

  • Autoimmune Hypophysitis

    All 4 PH and 4 out of 8 INH were treated with glucocorticoids.[] The patients were treated with glucocorticoids plus azathioprine, and positive responses were seen in all three cases.[] Early initiation of an intravenous dose of glucocorticoid followed by oral steroid administration therefore seems to be an efficient treatment for autoimmune hypophysitis[]

  • Osteoporosis

    Alternate-day glucocorticoid therapy can lead to similar bone loss. No conclusive evidence exists for a safe minimum dose or duration of glucocorticoid exposure.[] Abstract Objective To investigate the efficacy of minodronate in the treatment of glucocorticoid-induced osteoporosis (GIO).[] BACKGROUND: Zoledronic acid (ZOL) is widely used for treatment of glucocorticoid-induced osteoporosis.[]

  • Iatrogenic Cushing's Disease

    […] not prescribed glucocorticoids (table 5).[] Abbreviations: ACTH — adrenocorticotropic hormone CS — Cushing’s syndrome GC — glucocorticoid GR — glucocorticoid receptor SNP — single-nucleotide polymorphism 3′ UTR — 3′[] […] with the group not prescribed glucocorticoids.[]

  • Hyperaldosteronism

    Abstract Glucocorticoid-suppressible hyperaldosteronism is a dominantly inherited form of hypertension believed to be caused by the presence of a hybrid CYP11B1/CYP11B2 gene[] Biochemical abnormalities were now consistent with glucocorticoid excess without hyperaldosteronism. She died four months later.[] FH type I was ruled out by glucocorticoid suppression test, PCR test for CYP11B1/CYP11B2 gene, and urinary 18-oxo-cortisol and 18-hydroxy-cortisol excretion, which was in[]

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