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87 Possible Causes for Hyperreninemia

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  • Gitelman Syndrome

    The main features of this syndrome include normal or low blood pressure, hypokalemia, metabolic alkalosis, hypomagnesemia, hypocalciuria, and hyperreninemia.[] Laboratory blood tests disclosed hypokalemia, metabolic alkalosis, hyperreninemia, and hyperaldosteronism. Plasma magnesium concentration was slightly low.[] Both probands show hypokalemia, hypochloraemia, hypocalcinuria, hyperreninemia, and hyperaldosteronemia; Proband A has normal serum magnesium and increased urinary sodium[]

  • Antenatal Bartter Syndrome

    Rofecoxib suppressed hyperreninemia to a similar extent as indomethacin.[] Bartter's syndrome is a group of inherited, salt-losing tubulopathies presenting as metabolic alkalosis with normotensive hyperreninemia and hyperaldosteronism.[] In patients with HPS/aBS, excessive PGE2 synthesis and hyperreninemia is dependent on COX-2 activity.[]

  • EAST Syndrome

    Clinical presentation of the patients was homogeneous and included premature birth attributable to polyhydramnios, severe renal salt loss, normotensive hyperreninemia, hypokalemic[]

  • Autosomal Dominant Hypocalcemia Type 1

    Few patients manifest hypocalcemia and features of Bartter syndrome, including hypomagnesemia, hypokalemia, metabolic alkalosis, hyperreninemia, and hyperaldosteronemia.[] Four activating mutations of the CaSR cause additional renal sodium, chloride and magnesium wasting which results in hyperreninemia, hyperaldosteronism, hypokalemia, and metabolic[] […] resembling the salt-wasting Bartter syndrome (also referred to as Bartter syndrome subtype V), characterized by hypocalcemia, hypomagnesemia, hypokalemia, metabolic alkalosis, hyperreninemia[]

  • Bartter's Disease

    Lab Metabolic alkalosis, decreased serum K , increased urine K , increased renin, increased aldosterone.[] Bartter syndrome and Gitelman syndrome are characterized by fluid, electrolyte, urinary, and hormonal abnormalities, including renal K, Na, Cl, and H wasting; hypokalemia; hyperreninemia[] Bartter syndrome Endocrinology A rare endocrinopathy, usually of younger Pts, and may be seen in dwarfism; it is characterized by potassium-wasting, normotensive hyperreninemia[]

  • Hypokalemic Nephropathy

    Excessive aldosterone production may result from congenital adrenal hyperplasia, glucocorticoid treatment, some genetic defects and hyperreninemia. • Intrinsic renal potassium[] On the other hand, sustained hypokalemia and hyperreninemia can cause progressive tubulointerstitial nephritis , resulting in end-stage kidney disease (kidney failure).[] […] blood pressure, the most often causes of hypokalemia, like excessive sweating, diarrhea, starvation, milk-alkali syndrome, drug-induced potassium losses, hyperaldosteronism, hyperreninemia[]

  • Hypoaldosteronism

    These results suggest adrenal insensitivity to angiotensin II, possibly a defect in adrenal angiotensin II receptors, as the cause of hypoldosteronism with hyperreninemia[] Case Report Abstract Author Information We report a child with primary renal tubular alkalosis who had hypokalemia , profound hyperreninemia , and hypoaldosteronism .[] Laboratory data revealed selective aldosterone deficiency with hyperreninemia.[]

  • Congenital Mesoblastic Nephroma

    In the present article, we report, for the first time, a prenatal diagnosis of a congenital mesoblastic nephroma in combination with a post-partum hyperreninemia with hypertension[] Polyhydramnios, with or without hydrops fetalis, and/or preterm labor are associated with CMN. [7] Some CMNs are associated with paraneoplastic syndromes such as hypertension (due to hyperreninemia[] Hyperreninemia and congenital mesoblastic nephroma: case report and review of the literature. Urology 2000;55:775. 6. Ko SM, Kim MJ, Im YJ, Park KI, Lee MJ.[]

  • Familial Isolated Hypoparathyroidism

    […] resembling the salt-wasting Bartter syndrome (also referred to as Bartter syndrome subtype V), characterized by hypocalcemia, hypomagnesemia, hypokalemia, metabolic alkalosis, hyperreninemia[]

  • Essential Hypertension

    Hyperreninemia promotes conversion of Ang I to Ang II, causing severe vasoconstriction and aldosterone release.[]

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