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839 Possible Causes for Hypocalcemia, Metabolic Acidosis

  • Ethylene Glycol

    Recurrent severe anion gap metabolic acidosis consequent to episodic ingestion of ethylene glycol has not been previously reported.[] One of these findings is hypocalcemia, presumable through complexation with the ethylene glycol metabolite oxalate.[] Renal failure, acidosis, hypocalcemia, and death may follow.[]

  • Renal Tubular Acidosis

    We diagnosed him with relapse of metabolic acidosis caused by renal tubular acidosis. [Indexed for MEDLINE] Free full text[] Here, as a unique coexistence of these two seperated clinical entities, we report a 30-yr-old woman presenting with carpal spasm related to hypocalcemia (ionized calcium of[] […] investigations revealed chronic kidney disease with serum creatinine of 2.3 mg/dL and creatinine clearance of 40 mL/min, hypokalemia (3.2 mmol/L), hypophosphatemia (0.4 mmol/L), hypocalcemia[]

  • Proximal Renal Tubular Acidosis

    Thus, NBC1(W516X/W516X) mice with pRTA represent an animal model for metabolic acidosis and may be useful for testing therapeutic inhibition of NMD in vivo.[] Normal anion gap metabolic acidosis/acidemia Hypokalemia, Hypocalcemia, Hyperchloremia Urinary stone formation (related to alkaline urine, hypercalciuria, and low urinary[] […] failure and in experimental metabolic acidosis in animals.[]

  • Distal Renal Tubular Acidosis

    […] after metabolic acidosis correction.[] Serum electrolytes showed hypocalcemia (7.2 mg/dl), hypophosphatemia (2.8 mg/dl) with normal serum magnesium and sodium.[] […] features : Gauibad-Vainsel syndrome Osteopetrosis Cerebral calcification Mental retardation Diagnostics Serum Hyperchloremic ( normal anion gap ) metabolic acidosis Hypokalemia Hypocalcemia[]

  • Acute Kidney Failure

    Medical treatment for hyperkalaemia and metabolic acidosis was initiated as well as plasma volume expansion.[] Hypocalcemia is thought to occur because the impaired kidney no longer produces calcitriol and because hyperphosphatemia causes calcium phosphate precipitation in the tissues[] The hypocalcemia associated with AKI is usually asymptomatic and does not require specific treatment.[]

  • Acute Pancreatitis

    Though the symptoms were rapidly relieved after initiation of treatment, severe hyperglycemia (575 mg/dL), severe metabolic acidosis (pH 6.9), and ketonuria developed at four[] The notion that these might be useful prognostic markers is based partially on the observation that metabolic acidosis (characterized by reduced pH, bicarbonate and base excess[] : Lipase breaks down peripancreatic and mesenteric fat release of free fatty acids that bind calcium hypocalcemia References: [5] [6] [7] Clinical features Constant, severe[]

  • Chronic Kidney Insufficiency

    While this is not an all-inclusive list, some of the common conditions are: Anemia Hypertension Electrolyte imbalance, hyperparathyroidism Metabolic acidosis and alkalosis[] Hypocalcemia (see chapter) Hyponatremia (see chapter) Uremia: The three most severe forms of uremia are encephalopathy, bleeding diathesis, and pericarditis.[] This metabolic acidosis leads to increased bone calcium release and strengthening renal osteopathy, more gastrointestinal problems and an impairment of protein metabolism.[]

  • Alkalosis

    What are the symptoms of metabolic acidosis and alkalosis? If you have metabolic acidosis, you may have no symptoms.[] Signs of hypocalcemia may also develop. Patients with chronic respiratory alkalosis do not usually exhibit symptoms.[] […] contractility Arrhythmias Neurological consequences Neuromuscular overexcitability Decreased cerebral blood flow due to cerebral vasoconstriction Seizures Electrolyte changes Hypocalcemia[]

  • Anorexia Nervosa

    However, measuring the serum 25(OH)D levels in AN patients with hypocalcemia is recommended to prevent osteomalacia and osteoporosis. [Indexed for MEDLINE] Free full text[] Nutritional support was begun slowly, however electrolyte derangements began to manifest on hospital day 2, with hypophosphatemia, hypokalemia, hypocalcemia, and hypomagnesemia[] Dehydration and prerenal azotemia are not uncommon; in addition, other electrolyte abnormalities such as hypocalcemia and hypomagnesemia can be present.[]

  • Tumor Lysis Syndrome

    After treatment, the patient developed acute tumor lysis syndrome with hyperkalemia, hyperuricemia, hyperphosphatemia, hypocalcemia, metabolic acidosis and acute renal failure[] Other signs are hyperkalemia, hyperphosphatemia and secondary hypocalcemia.[] Despite intensive hemodialysis and other supportive therapy, he developed multiple organ failure (liver, renal, and respiratory failure) and metabolic acidosis.[]

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