Fat necrosis was present. The possible pathophysiologic mechanisms are discussed.[ncbi.nlm.nih.gov]
In addition, both anaphylatoxin C5a and the terminal complement complex C5b-9 were present at high levels, comparable to those in RA patients.[ncbi.nlm.nih.gov]
This study describes two infants presenting with acute acalculous cholecystitis with marked distension of the gall bladder.[ncbi.nlm.nih.gov]
Although present knowledge is incomplete to delineate an accurate scenario and a schedule of the events occurring under magnesium deficiency, it emerges that low magnesium status favors the induction of acute inflammation by sensitizing sentinel cells[ncbi.nlm.nih.gov]
This review presents recent advances in understanding the formation and action of these mediators, especially focusing on the LC-MS/MS-based lipidomics approach and the emerging roles of eosinophils and eosinophil-derived lipid mediators in controlling[ncbi.nlm.nih.gov]
Taken together, we should be aware of the possibility of vildagliptin inducing drug fever and/or acute inflammation.[ncbi.nlm.nih.gov]
She suffered from diabetic foot gangrene in her right foot with high-grade fever when she visited our hospital.[ncbi.nlm.nih.gov]
It begins with sudden fever, which reaches, and sometimes exceeds 40C. The fever is accompanied by shivers and one- or both-side lumbar pains, which are sometimes very strong. Symptoms of acute inflammation of urinary bladder appear very often.[archive.ics.uci.edu]
She was immediately admitted to our hospital and received intensive insulin treatment, along with intravenous-administration of antibiotics.[ncbi.nlm.nih.gov]
The necropsy findings in an elderly woman, who had been treated with methyldopa and who had died during treatment for accidental hypothermia, showed acute periductal inflammation of obviously similar duration in an entopic pancreas in the wall of the[ncbi.nlm.nih.gov]
Interleukin-6increased (P 0.05) 8 h postexercise, with a trend (P 0.09) to be greater in the overweight group. CRP and cortisol responses were not detected.[ncbi.nlm.nih.gov]
Inadequate and delayed decisions makes acute inflammation the 10th leading cause of death overall in United States with the estimated cost of treatment about 17 billion annually.[ncbi.nlm.nih.gov]
The necropsy findings in an elderly woman, who had been treated with methyldopa and who had died during treatment for accidental hypothermia, showed acute periductal inflammation of obviously similar duration in an entopic pancreas in the wall of the[ncbi.nlm.nih.gov]
Treatment with lipopolysaccharide (LPS) stimulated retinal Angptl2 mRNA expression in vivo and in vitro. We generated EIU in wild-type (C57BL/6) and Angptl2 / mice by injecting LPS intraperitoneally.[ncbi.nlm.nih.gov]
Our study provides the essential information for better understanding the molecular mechanisms of CDI and the potential new therapeutic strategies for the treatment of this infection. The Author 2016.[ncbi.nlm.nih.gov]
We examined various kinds of cytokine and infection markers just before and after the treatment with vildagliptin.[ncbi.nlm.nih.gov]
Depending on the etiology, localization and composition, exudative inflammation can be : serous (first stage of lobar pneumonia, vesicles in herpetic infections or burns) fibrinous (second stage of lobar pneumonia, fibrinous pericarditis, peritonitis[pathologyatlas.ro]
Animals Biglycan/genetics Biglycan/metabolism Blotting, Western Collagen Type I/genetics* Collagen Type I/metabolism Diet, High-Fat Energy Intake Extracellular Matrix Proteins/genetics Extracellular Matrix Proteins/metabolism Female Humans Inflammation/etiology[ncbi.nlm.nih.gov]
The possible pathophysiologic mechanisms are discussed.[ncbi.nlm.nih.gov]
Previous studies on EGC pathophysiology have specifically focused on mucosal glia activation and its contribution to mucosal inflammatory processes observed in the gut of inflammatory bowel disease (IBD) patients.[ncbi.nlm.nih.gov]
Despite the prevalence and severity of sepsis, we lack a fundamental grasp of its pathophysiology. Here we report that the cytokine interleukin-3 (IL-3) potentiates inflammation in sepsis.[ncbi.nlm.nih.gov]
Author information 1 Tianjin Key Laboratory on Technologies Enabling Development of Clinical Therapeutics and Diagnostics, School of Pharmacy, Tianjin Medical University, Tianjin, 300070, PR China. 2 Department of Physiology & Pathophysiology, Tianjin[ncbi.nlm.nih.gov]
: Defect in integrin b 2 chain (shared by LFA-1 and Mac-1): CD18 Leukocyte Adhesion Deficiency Type II Clinical: Impaired leukocyte adhesion and recurrent bacterial infections (milder than LAD1) Pathophysiology: Absence of sialyl-Lewis X, ligand for E-selectin[pathologyexpert.com]
To limit inflammation and prevent collateral injury of healthy, uninvolved tissue, the lung orchestrates the formation of specialized proresolving mediators, specifically lipoxins, resolvins, protectins, and maresins.[ncbi.nlm.nih.gov]
Although myeloid cell activation is requisite for an optimal innate immune response, this process must be tightly controlled to prevent collateral host tissue damage.[ncbi.nlm.nih.gov]
In murine models of LPS- and Con A-induced liver injury, we found that naringenin, a natural predominant flavanone, is capable of protecting against lethality induced by LPS and preventing inflammation-induced organ injury.[ncbi.nlm.nih.gov]
Despite improvements in early detection, prevention, and treatment, the mortality rate in breast cancer remains high secondary to the potential for cancer recurrence and the development of metastasis.[ncbi.nlm.nih.gov]
Therefore, the local inhibition of specific prostanoid receptor signaling is expected to be an effective strategy for the prevention and treatment of acute inflammation. Copyright 2014 Elsevier Masson SAS. All rights reserved.[ncbi.nlm.nih.gov]