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Rhus Verniciflua

Japanese Lacquer Tree


Presentation

  • PATIENT AND METHOD: We present here the case of a female patient (82 years old) with an adenocarcinoma of the stomach that was first diagnosed via an abdomen computed tomography (CT) scan and endoscopic biopsy.[ncbi.nlm.nih.gov]
  • In the present study, three different allergen-free R. verniciflua extracts (DRVE, FRVE, and FFRVE) were produced by detoxification of R. verniciflua, and their properties and constituents were compared.[ncbi.nlm.nih.gov]
  • OBJECTIVES: The present study investigated the neuroprotective effects of Rhus verniciflua Stokes (RVS) leaf extract on rotenone-induced apoptosis in human dopaminergic cells, SH-SY5Y.[ncbi.nlm.nih.gov]
  • In the present study, we prepared an extract of the allergen‑removed RVS (aRVS) based on a traditional method and investigated its inhibitory effect on the growth of various types of human cancer cells, including lung (A549), breast (MCF-7) and prostate[ncbi.nlm.nih.gov]
  • In the present study, fermented R. verniciflua Stokes extract (FRVE), an allergen-free extract of R. verniciflua Stokes fermented with the yeast Saccharomyces carlsbergensis, was assessed for its lipid-lowering potential in an in vitro non-alcoholic fatty[ncbi.nlm.nih.gov]
Epigastric Pain
  • Patients who had better performance status and adenocarcinoma experienced more favorable outcomes in terms of overall survival. in aRVS treatment were negligible, with the most common drug-related adverse events being mild epigastric pain and itching[ncbi.nlm.nih.gov]
Formication
  • The chromatographic separation was performed using a C18 column under gradient elution with 0.1% formic acid and acetonitrile as the mobile phase solvents.[ncbi.nlm.nih.gov]
  • The mobile phase consisted of water containing 0.1% formic acid (solvent A) and acetonitrile containing 0.1% formic acid (solvent B). The gradient elution was 0–5 min, 5% B, 5–30 min, 5–95% B.[journals.plos.org]
  • The mobile phases were composed of 0.1% formic acid in water (solvent A) and 100% methanol (solvent B), delivered at a flow rate of 0.7 mL/min.[bmccomplementalternmed.biomedcentral.com]
Suggestibility
  • Collectively, these findings suggest that RCMF is an agent which may be capable of inducing sensitive growth inhibition and apoptosis in HOS cells.[ncbi.nlm.nih.gov]
  • BACKGROUND: Many studies have suggested that the flavonoids from Rhus verniciflua Stokes (RVS) are anticancer agents, but a few clinical studies have reported on this topic.[ncbi.nlm.nih.gov]
  • Taken together, these results suggest that RVS modulates the expressions of signal molecules related to allergic inflammatory responses mainly through the ERK signaling pathway, suggesting that RVS could be used as a treatment for mast cell-derived allergic[ncbi.nlm.nih.gov]
  • Since aflatoxin B(1) (AFB(1))-mediated hepatic damage is related to the production of AFB(1)-8,9-epoxide and reactive oxygen species, bioactive compounds having antioxidant potentials are suggested to be capable of reducing AFB(1)-induced toxicity.[ncbi.nlm.nih.gov]
  • CONCLUSIONS: These findings demonstrate that RVS protects SH-SY5Y cells against rotenone-induced injury and suggest that RVS may have potential therapeutic value for neurodegenerative disease associated with oxidative stress. 2011 The Authors.[ncbi.nlm.nih.gov]
Stroke
  • We investigated the mechanisms involved in mediating the effects of Rhus verniciflua Strokes (R. verniciflua) extract in human chronic myelogenous leukemia K562 cells, including caspase-dependent apoptotic pathways related to cell-cycle arrest, as well[ncbi.nlm.nih.gov]
  • Click on the brush button to enter characters just by drawing them with the correct stroke order. Stroke order animation. Sample .[yellowbridge.com]
  • The phenylpropanoid micronutrient chlorogenic acid improves clinical rating scores in rabbits following multiple infarct ischemic strokes: synergism with tissue plasminogen activator. Exp Neurol 2007;205:407–413. 27.[synapse.koreamed.org]

Treatment

  • Furthermore, aRVS extract treatment largely reduced the survival and induced apoptosis of the A549 cells.[ncbi.nlm.nih.gov]
  • BACKGROUND: Systemic treatments for advanced non-small cell lung cancer (NSCLC) have modest survival benefits but high toxicity.[ncbi.nlm.nih.gov]
  • Traditionally, this remedy has been used in Eastern Asia for the treatment of gastric problems, hepatic disorders, infectious diseases, and blood disorders.[ncbi.nlm.nih.gov]
  • Moreover, combined treatment of an ethanol extract of RVS and LY294002 (a PI3K inhibitor) markedly increased apoptosis compared to treatment with an ethanol extract of RVS alone.[ncbi.nlm.nih.gov]
  • Adverse reactions to the RVS treatment were mostly mild and self-limiting.[ncbi.nlm.nih.gov]

Prognosis

  • BACKGROUND: Pancreatic cancer has the worst prognosis because of poor response to conventional therapy.[ncbi.nlm.nih.gov]
  • December 14, 2011 Issue release date: February 2012 Number of Print Pages: 7 Number of Figures: 2 Number of Tables: 4 ISSN: 0030-2414 (Print) eISSN: 1423-0232 (Online) For additional information: Abstract Background: Pancreatic cancer has the worst prognosis[karger.com]

Pathophysiology

  • Glial cell reactions in neurodegenerative diseases: pathophysiology and therapeutic interventions. Alzheimer Dis Assoc Disord 1998;12 Suppl 2:S1–S6. 5. Itagaki S, McGeer PL, Akiyama H, Zhu S, Selkoe D.[synapse.koreamed.org]

Prevention

  • Abstract Despite the fact that numerous researches were performed on prevention and treatment of inflammation related diseases, the overall incidence has not changed remarkably.[ncbi.nlm.nih.gov]
  • Although 6-OHDA-increased phosphorylation of p38 was prevented by NAC or BAPTA, inhibition of p38 by SB203580 did not suppress ROS, Bax/Bcl-2 ratio, or caspase-3 activity increases, and only partially prevented 6-OHDA-induced cell death, thus demonstrating[ncbi.nlm.nih.gov]
  • RVS prevented cisplatin-induced cytotoxicity and ROS release against MDCK-I cells. RVS alone exerted modest antitumor activity against CT-26 cells.[ncbi.nlm.nih.gov]
  • RCMF also prevented the reduction in HepG2 cell viability caused by AFB(1).[ncbi.nlm.nih.gov]
  • Moreover, pretreatment with RVS prevented the decrease in tyrosine hydroxylase (TH) levels in SH-SY5Y cells.[ncbi.nlm.nih.gov]

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