Reciprocal expression of interferon gamma or interleukin 4 during the resolution or progression of murine leishmaniasis. Evidence for expansion of distinct helper T cell subsets.
Leishmaniasis worldwide and global estimates of its incidence.
WHO Leishmaniasis Control Team;
Advances in leishmaniasis.
2005 Oct 29-Nov 4:
Heterogeneities in the transmission of infectious agents: implications for the design of control programs.
Drug resistance in leishmaniasis.
Immunoregulation of cutaneous leishmaniasis. T cell lines that transfer protective immunity or exacerbation belong to different T helper subsets and respond to distinct parasite antigens.
Macrophage killing of Leishmania parasite in vivo is mediated by nitric oxide from L-arginine.
Comparative genomic analysis of three Leishmania species that cause diverse human disease.
Cure of murine leishmaniasis with anti-interleukin 4 monoclonal antibody. Evidence for a T cell-dependent, interferon gamma-independent mechanism.
Lymphocytes bearing antigen-specific gamma delta T-cell receptors accumulate in human infectious disease lesions.
SOCS1 is a critical inhibitor of interferon gamma signaling and prevents the potentially fatal neonatal actions of this cytokine.
Production of interferon gamma, interleukin 2, interleukin 4, and interleukin 10 by CD4+ lymphocytes in vivo during healing and progressive murine leishmaniasis.
A limiting dilution assay for quantifying Leishmania major in tissues of infected mice.
The relationship between leishmaniasis and AIDS: the second 10 years.
Leishmania major amastigotes initiate the L-arginine-dependent killing mechanism in IFN-gamma-stimulated macrophages by induction of tumor necrosis factor-alpha.
Identification of an infective stage of Leishmania promastigotes.
Tumor necrosis factor-alpha synergizes with IFN-gamma in mediating killing of Leishmania major through the induction of nitric oxide.
The increase in risk factors for leishmaniasis worldwide.
Administration of monoclonal anti-IFN-gamma antibodies in vivo abrogates natural resistance of C3H/HeN mice to infection with Leishmania major.
PHvan der Meide;