The hallmarks of cancer.
Tumor angiogenesis: therapeutic implications.
Establishment in culture of pluripotential cells from mouse embryos.
Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumours.
In vivo activation of the p53 pathway by small-molecule antagonists of MDM2.
Transfer of proteins across membranes. I. Presence of proteolytically processed and unprocessed nascent immunoglobulin light chains on membrane-bound ribosomes of murine myeloma.
Isolation of a pluripotent cell line from early mouse embryos cultured in medium conditioned by teratocarcinoma stem cells.
Stat3 as an oncogene.
Heterozygous embryonic lethality induced by targeted inactivation of the VEGF gene.
Tumor spectrum analysis in p53-mutant mice.
A dye-buoyant-density method for the detection and isolation of closed circular duplex DNA: the closed circular DNA in HeLa cells.
Nitric oxide and macrophage function.
Endostatin: an endogenous inhibitor of angiogenesis and tumor growth.
A new concept for macromolecular therapeutics in cancer chemotherapy: mechanism of tumoritropic accumulation of proteins and the antitumor agent smancs.
Tumor cells secrete a vascular permeability factor that promotes accumulation of ascites fluid.
Biology of natural killer cells.
Inhibition of vascular endothelial growth factor-induced angiogenesis suppresses tumour growth in vivo.
Mice lacking p21CIP1/WAF1 undergo normal development, but are defective in G1 checkpoint control.
Telomere shortening and tumor formation by mouse cells lacking telomerase RNA.