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490801 to 490900 most common queries
List represents a sample of symptoms, diseases, and other queries. Updated weekly.
Linkage relationship of a cloned DNA sequence on the short arm of the X chromosome to Duchenne muscular dystrophy.
The molecular basis of visual excitation.
Molecular basis of visual excitation.
Molecular genetics of inherited variation in human color vision.
anemia, fatigue, bone pain, renal blood, fatty liver, weight gain, benign nodules , jaundice, leukocytosis, fever, pallor, chronic pain, liver tenderness, skin pigmentation, edema
In search of a selective antiviral chemotherapy.
Characterization of age- and dose-related outcomes of duck hepatitis B virus infection.
John Richard Graybill
Transfer of hepatitis B virus genome by adenovirus vectors into cultured cells and mice: crossing the species barrier.
A new avian hepadnavirus infecting snow geese (Anser caerulescens) produces a significant fraction of virions containing single-stranded DNA.
Single-cell analysis of covalently closed circular DNA copy numbers in a hepadnavirus-infected liver.
anemia, fatigue, bone pain, renal blood, fatty liver, weight gain, benign nodules , jaundice, leukocytosis, fever, pallor, chronic pain, liver tenderness, skin pigmentation
In vitro model for the nuclear transport of the hepadnavirus genome.
Susceptibility to duck hepatitis B virus infection is associated with the presence of cell surface receptor sites that efficiently bind viral particles.
brain fog, fatigue, chronic tension headache,
Evidence that hepatocyte turnover is required for rapid clearance of duck hepatitis B virus during antiviral therapy of chronically infected ducks.
A new group of hepadnaviruses naturally infecting orangutans (Pongo pygmaeus).
Duck hepatitis B virus: an invaluable model system for HBV infection.
A splice hepadnavirus RNA that is essential for virus replication.
anemia, fatigue, bone pain, renal blood, fatty liver, weight gain, benign nodules , jaundice, leukocytosis, fever, pallor, chronic pain, liver tenderness
Low dynamic state of viral competition in a chronic avian hepadnavirus infection.
Avian hepatitis B virus infection is initiated by the interaction of a distinct pre-S subdomain with the cellular receptor gp180.
Occult lifelong persistence of infectious hepadnavirus and residual liver inflammation in woodchucks convalescent from acute viral hepatitis.
Molecular events in the pathogenesis of hepadnavirus-associated hepatocellular carcinoma.
The half-life of duck hepatitis B virus supercoiled DNA in congenitally infected primary hepatocyte cultures.
Hepatocyte turnover during resolution of a transient hepadnaviral infection.
Kinetics of duck hepatitis B virus infection following low dose virus inoculation: one virus DNA genome is infectious in neonatal ducks.
anemia, fatigue, bone pain, renal blood, fatty liver, weight gain, benign nodules , jaundice, leukocytosis, fever, pallor, chronic pain
Hepadnavirus infection requires interaction between the viral pre-S domain and a specific hepatocellular receptor.
Viral and cellular determinants involved in hepadnaviral entry.
Comparison of preseptal and pretarsal injections of botulinum toxin in the treatment of blepharospasm and hemifacial spasm.
Botulinum toxin A treatment for primary hemifacial spasm: a 10-year multicenter study.
Beneficial effects of botulinum toxin type a for patients with painful tic convulsif.
An attempt to relieve hemifacial spasm by neurolysis of the facial nerves; a report of two cases of hemifacial spasm with reflections on the nature of the spasm, the contracture and mass movement.
Review of the FDA-approved uses of botulinum toxins, including data suggesting efficacy in pain reduction.
Hemifacial spasm and involuntary facial movements.
DYSBOT: a single-blind, randomized parallel study to determine whether any differences can be detected in the efficacy and tolerability of two formulations of botulinum toxin type A--Dysport and Botox--assuming a ratio of 4:1.
The many faces of hemifacial spasm: differential diagnosis of unilateral facial spasms.
anemia, fatigue, bone pain, renal blood, fatty liver, weight gain, benign nodules , jaundice, leukocytosis, fever, pallor
Psychiatric disorders in adult-onset focal dystonia: a case-control study.
Prognostic value of the lateral spread response for intraoperative electromyography monitoring of the facial musculature during microvascular decompression for hemifacial spasm.
Long-term botulinum toxin efficacy, safety, and immunogenicity.
Health-related quality of life and psychosocial characteristics of patients with benign essential blepharospasm.
Tactile temporal discrimination in patients with blepharospasm.
Abnormal perception of vibration-induced illusion of movement in dystonia.
Hemifacial spasm: clinical findings and treatment.
Abnormal plasticity of sensorimotor circuits extends beyond the affected body part in focal dystonia.
Compression syndromes caused by substernal goitres.
Acute respiratory failure and tracheal obstruction in patients with intrathoracic goiter.
Surgical management of substernal goitre.
Surgical management of substernal goiters: clinical experience of 170 cases.
Surgical aspects of 175 mediastinal goiters.
Morbidity and mortality of thyroidectomy for substernal goiter.
anemia, fatigue, bone pain, renal blood, fatty liver, weight gain, benign nodules , jaundice, leukocytosis, fever
Low-attenuation mediastinal masses on CT.
Substernal goiter: a clinical review.
Parathyroid preservation during thyroid surgery.
Goiters and airway problems.
Benign superior vena cava syndrome.
Rationale for the operative management of substernal goiters.
EXPERIENCE WITH MEDIASTINOSCOPY.
Thyroidectomy in patients with marked thyroid enlargement: airway management, morbidity, and outcome.
Evidence-based surgical management of substernal goiter.
Increasing incidence of thyroid cancer is due to increased pathologic detection.
Primary mediastinal tumors. Part 1: tumors of the anterior mediastinum.
Vestibulocochlear Nerve Injuries
Hematoma, Epidural, Spinal
Deep brain stimulation in chorea acanthocytosis.
Chorein-sensitive polymerization of cortical actin and suicidal cell death in chorea-acanthocytosis.
Computational identification of phospho-tyrosine sub-networks related to acanthocyte generation in neuroacanthocytosis.
A neuropathological study of autosomal-dominant chorea-acanthocytosis with a mutation of VPS13A.
Cardiac and neurologic involvement in McLeod syndrome.
Novel pathogenic mutations and copy number variations in the VPS13A gene in patients with chorea-acanthocytosis.
Identification and characterization of a novel XK splice site mutation in a patient with McLeod syndrome.
Evolution of striatal degeneration in McLeod syndrome.
Development of mesial temporal lobe epilepsy in chorea-acanthocytosis.
Characteristic head drops and axial extension in advanced chorea-acanthocytosis.
Cardiac abnormalities in McLeod syndrome.
Automatic striatal volumetry allows for identification of patients with chorea-acanthocytosis at single subject level.
McLeod myopathy revisited: more neurogenic and less benign.
Brain, blood, and iron: perspectives on the roles of erythrocytes and iron in neurodegeneration.
Tongue protrusion and feeding dystonia: a hallmark of chorea-acanthocytosis.
Shape alterations in the striatum in chorea-acanthocytosis.
Erythrocyte membrane changes of chorea-acanthocytosis are the result of altered Lyn kinase activity.
McLeod syndrome: a neurohaematological disorder.
Genetic diagnosis of neuroacanthocytosis disorders using exome sequencing.
Pulmonary Adenomatosis, Ovine
KIT mutation in mast cells and other bone marrow hematopoietic cell lineages in systemic mast cell disorders: a prospective study of the Spanish Network on Mastocytosis (REMA) in a series of 113 patients.
Tryptase levels as an indicator of mast-cell activation in systemic anaphylaxis and mastocytosis.
Mid-Face Hypoplasia, Short Neck, Short Stature in Children
Antibodies to IL-3 and IL-4 suppress helminth-induced intestinal mastocytosis.
Activating and dominant inactivating c-KIT catalytic domain mutations in distinct clinical forms of human mastocytosis.
Standards and standardization in mastocytosis: consensus statements on diagnostics, treatment recommendations and response criteria.
The c-KIT mutation causing human mastocytosis is resistant to STI571 and other KIT kinase inhibitors; kinases with enzymatic site mutations show different inhibitor sensitivity profiles than wild-type kinases and those with regulatory-type mutations.