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505101 to 505200 most common queries
List represents a sample of symptoms, diseases, and other queries. Updated weekly.
Neurovisceral porphyrias: what a hematologist needs to know.
Digenic inheritance of mutations in the coproporphyrinogen oxidase and protoporphyrinogen oxidase genes in a unique type of porphyria.
Coproporphyrinuria in groups of workers as an index of inorganic lead absorption.
Coproporphyrinuria as an index of lead absorption.
The incidence of abnormal blood levels of lead in a metropolitan pediatric clinic; with observation on the value of coproporphyrinuria as a screening test.
The acute hepatic porphyrias: current status and future challenges.
Dual gene defects involving delta-aminolaevulinate dehydratase and coproporphyrinogen oxidase in a porphyria patient.
Mutations in human CPO gene predict clinical expression of either hepatic hereditary coproporphyria or erythropoietic harderoporphyria.
Role of porphyrins in occupational diseases; significance of coproporphyrinuria in lead workers.
Structural basis of hereditary coproporphyria.
Modern diagnosis and management of the porphyrias.
Nasopharyngeal angiofibromas: selecting a surgical approach.
Imaging for juvenile angiofibroma.
Annotation: tuberous sclerosis.
Successful treatment of angiofibromata of tuberous sclerosis complex with rapamycin.
Cellular angiofibroma: a benign neoplasm distinct from angiomyofibroblastoma and spindle cell lipoma.
Dramatic improvement of facial angiofibromas in tuberous sclerosis with topical rapamycin: optimizing a treatment protocol.
Juvenile angiofibroma: the lessons of 20 years of modern imaging.
Complete inactivation of the TSC2 gene leads to formation of hamartomas.
The results of surgery for benign tumors of the cavernous sinus.
Pulmonary lymphangioleiomyomatosis in a man.
Cutaneous tumors in patients with multiple endocrine neoplasia type 1 show allelic deletion of the MEN1 gene.
Loss of tuberin, the tuberous-sclerosis-complex-2 gene product is associated with angiogenesis.
The mTOR inhibitor rapamycin significantly improves facial angiofibroma lesions in a patient with tuberous sclerosis.
Tuberous sclerosis-associated lesions of the kidney, brain, and skin are angiogenic neoplasms.
Multiple endocrine neoplasia type 1: new clinical and basic findings.
Loss of heterozygosity at 11q13: analysis of pituitary tumors, lung carcinoids, lipomas, and other uncommon tumors in subjects with familial multiple endocrine neoplasia type 1.
Tuberous sclerosis complex consensus conference: revised clinical diagnostic criteria.
Evaluation of the antitumor activity of gemcitabine (2',2'-difluoro-2'-deoxycytidine).
Murine cutaneous leishmaniasis: resistance correlates with the capacity to generate interferon-gamma in response to Leishmania antigens in vitro.
Hypertrophic scarring and keloids: pathomechanisms and current and emerging treatment strategies.
Hic-5 promotes the hypertrophic scar myofibroblast phenotype by regulating the TGF-beta1 autocrine loop.
The dual-specific binding of dengue virus and target cells for the antibody-dependent enhancement of dengue virus infection.
Histoplasma capsulatum modulates the acidification of phagolysosomes.
Focal adhesion kinase links mechanical force to skin fibrosis via inflammatory signaling.
Purification of topoisomerase II from amsacrine-resistant P388 leukemia cells. Evidence for two forms of the enzyme.
Mechanisms of pathological scarring: role of myofibroblasts and current developments.
Hypertrophic scar formation following burns and trauma: new approaches to treatment.
Mechanisms of oxidant-mediated cell injury. The glycolytic and mitochondrial pathways of ADP phosphorylation are major intracellular targets inactivated by hydrogen peroxide.
Ca(2+)-independent cytosolic phospholipase A2 from macrophage-like P388D1 cells. Isolation and characterization.
Histology of the thick scar on the female, red Duroc pig: final similarities to human hypertrophic scar.
Oxidant injury of cells. DNA strand-breaks activate polyadenosine diphosphate-ribose polymerase and lead to depletion of nicotinamide adenine dinucleotide.
Enhanced expression of transforming growth factor-beta type I and type II receptors in wound granulation tissue and hypertrophic scar.
Interaction of Listeria monocytogenes with human brain microvascular endothelial cells: InlB-dependent invasion, long-term intracellular growth, and spread from macrophages to endothelial cells.
Potential cellular and molecular causes of hypertrophic scar formation.
In vitro and in vivo reversal of multidrug resistance by GF120918, an acridonecarboxamide derivative.
Hypertrophic scars, keloids, and contractures. The cellular and molecular basis for therapy.
Pericytes as collagen-producing cells in excessive dermal scarring.
Cutting edge: receptor-mediated endocytosis of heat shock proteins by professional antigen-presenting cells.
squeezing pressure tension headache,with burning crawling sensations on scalp
The molecular basis of keloid and hypertrophic scar formation.
Intracellular roles of SN-38, a metabolite of the camptothecin derivative CPT-11, in the antitumor effect of CPT-11.
Increased accumulation of vincristine and adriamycin in drug-resistant P388 tumor cells following incubation with calcium antagonists and calmodulin inhibitors.
GM-CSF and oncogene mRNA stabilities are independently regulated in trans in a mouse monocytic tumor.
Mechanical load initiates hypertrophic scar formation through decreased cellular apoptosis.
Interleukin-induced increase in Ia expression by normal mouse B cells.
Inhibition of macrophage Ca(2+)-independent phospholipase A2 by bromoenol lactone and trifluoromethyl ketones.
International clinical recommendations on scar management.
Upregulation of the Wnt/beta-catenin pathway induced by transforming growth factor-beta in hypertrophic scars and keloids.
The beta-glucan receptor, dectin-1, is predominantly expressed on the surface of cells of the monocyte/macrophage and neutrophil lineages.
On the nature of hypertrophic scars and keloids: a review.
Bone marrow-derived stem cells in wound healing: a review.
Macrophages in skin injury and repair.
Expression of programmed death 1 ligands by murine T cells and APC.
Morphological and immunochemical differences between keloid and hypertrophic scar.
Cloning and expression of murine interleukin-1 cDNA in Escherichia coli.
Physiology, pharmacology and plasticity at the inner hair cell synaptic complex.
Guidelines for screening for hearing impairment and middle-ear disorders. Working Group on Acoustic Immittance Measurements and the Committee on Audiologic Evaluation. American Speech-Language-Hearing Association.
Wideband energy reflectance measurements in adults with middle-ear disorders.
Genetic factors in aminoglycoside toxicity.
Clinical, experimental, and theoretical investigations of the effect of superior semicircular canal dehiscence on hearing mechanisms.
Mice overexpressing genes from the 22q11 region deleted in velo-cardio-facial syndrome/DiGeorge syndrome have middle and inner ear defects.
Marine waters contaminated with domestic sewage: nonenteric illnesses associated with bather exposure in the United Kingdom.
Single or multiple daily doses of aminoglycosides: a meta-analysis.
Antimicrobial peptide therapeutics for cystic fibrosis.
Untoward effects associated with practolol administration: oculomucocutaneous syndrome.
A neurophysiological approach to tinnitus: clinical implications.
Aminoglycoside toxicity - a review of clinical studies published between 1975 and 1982.
Enhanced Leishmania braziliensis infection following pre-exposure to sandfly saliva.
Organic cation transporter 2 mediates cisplatin-induced oto- and nephrotoxicity and is a target for protective interventions.
Reconstitution of the rat olfactory epithelium after methyl bromide-induced lesion.
New hair cells arise from supporting cell conversion in the acoustically damaged chick inner ear.
Connexin disorders of the ear, skin, and lens.
Nuclear-factor kappa B (NF-kappa B)-inducible nitric oxide synthase (iNOS/NOS II) pathway damages the stria vascularis in cisplatin-treated mice.
Myo15 function is distinct from Myo6, Myo7a and pirouette genes in development of cochlear stereocilia.
Age-related cochlear synaptopathy: an early-onset contributor to auditory functional decline.
Hearing loss following Gata3 haploinsufficiency is caused by cochlear disorder.
Effect of protective agents against cisplatin ototoxicity.
The perception of temporal modulations by cochlear implant patients.
Glycine immunoreactivity and receptor binding in the cochlear nucleus of C57BL/6J and CBA/CaJ mice: effects of cochlear impairment and aging.
Significance of spiral ligament fibrocytes with cochlear inflammation.
squeezing pressure tension headache
Selective cochlear degeneration in mice lacking the F-box protein, Fbx2, a glycoprotein-specific ubiquitin ligase subunit.
p27(Kip1) deficiency causes organ of Corti pathology and hearing loss.
Cortical reorganization in patients with high frequency cochlear hearing loss.
Developmental delays consistent with cochlear hypothyroidism contribute to failure to develop hearing in mice lacking Slc26a4/pendrin expression.
A cochlear cell line as an in vitro system for drug ototoxicity screening.