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516801 to 516900 most common queries
List represents a sample of symptoms, diseases, and other queries. Updated weekly.
Methylprednisolone, valacyclovir, or the combination for vestibular neuritis.
Cerebellar infarction presenting isolated vertigo: frequency and vascular topographical patterns.
Functional brain imaging of peripheral and central vestibular disorders.
Phantom percepts: tinnitus and pain as persisting aversive memory networks.
Activity in the dorsal cochlear nucleus of hamsters previously tested for tinnitus following intense tone exposure.
Tinnitus--a study of its prevalence and characteristics.
Tinnitus and inferior colliculus activity in chinchillas related to three distinct patterns of cochlear trauma.
Salicylate induced tinnitus: behavioral measures and neural activity in auditory cortex of awake rats.
Plasticity at glycinergic synapses in dorsal cochlear nucleus of rats with behavioral evidence of tinnitus.
Reversing pathological neural activity using targeted plasticity.
Changes in spontaneous neural activity immediately after an acoustic trauma: implications for neural correlates of tinnitus.
Tuning out the noise: limbic-auditory interactions in tinnitus.
Ringing ears: the neuroscience of tinnitus.
Gap detection deficits in rats with tinnitus: a potential novel screening tool.
Development of the Tinnitus Handicap Inventory.
The effects of salsalate on glycemic control in patients with type 2 diabetes: a randomized trial.
The functional neuroanatomy of tinnitus: evidence for limbic system links and neural plasticity.
Reorganization of auditory cortex in tinnitus.
Phantom auditory perception (tinnitus): mechanisms of generation and perception.
Elevated fusiform cell activity in the dorsal cochlear nucleus of chinchillas with psychophysical evidence of tinnitus.
The neuroscience of tinnitus.
Thalamocortical dysrhythmia: A neurological and neuropsychiatric syndrome characterized by magnetoencephalography.
Lateral semicircular canal benign paroxysmal positional vertigo diagnostic signs.
Damaged utricular function clarified by oVEMP in patients with benign paroxysmal positional vertigo.
Comorbid benign paroxysmal positional vertigo in idiopathic sudden sensorineural hearing loss: an ominous sign for hearing recovery.
Clinical features of recurrent or persistent benign paroxysmal positional vertigo.
Clinical characteristics and treatment of benign paroxysmal positional vertigo after traumatic brain injury.
Classics from the laryngoscope.
Benign paroxysmal vertigo of childhood: long-term outcome.
Benign paroxysmal positional vertigo is a common cause of dizziness and unsteadiness in a large population of 75-year-olds.
Benign paroxysmal positional vertigo in the elderly.
Abnormality of cervical vestibular-evoked myogenic potentials and ocular vestibular-evoked myogenic potentials in patients with recurrent benign paroxysmal postitional vertigo.
[Benign paroxysmal positioning vertigo related to inner ear disorders].
Natural course of positional down-beating nystagmus of peripheral origin.
Modifications of the Epley (canalith repositioning) manoeuvre for posterior canal benign paroxysmal positional vertigo (BPPV).
Double-blind randomized trial on short-term efficacy of the Semont maneuver for the treatment of posterior canal benign paroxysmal positional vertigo.
Horizontal semicircular canal benign paroxysmal positional vertigo: effectiveness of two different methods of treatment.
Recurrence of benign paroxysmal positional vertigo.
Clinical characteristics of positional vertigo after mild head trauma.
Nystagmus assessments documented by emergency physicians in acute dizziness presentations: a target for decision support?
Sitting-up vertigo and trunk retropulsion in patients with benign positional vertigo but without positional nystagmus.
BENIGN PAROXYSMAL VERTIGO OF CHILDHOOD. (A VARIETY OF VESTIBULAR NEURONITIS).
Genetic heterogeneity in Rubinstein-Taybi syndrome: delineation of the phenotype of the first patients carrying mutations in EP300.
GIANT PIGMENTED NEVI, MELANOMA, AND LEPTOMENINGEAL MELANOCYTOSIS: A CLINICAL AND HISTOPATHOLOGICAL STUDY.
"Mistakes happen": somatic mutation and disease.
Neurovascular manifestations of heritable connective tissue disorders. A review.
Familial schwannomatosis: exclusion of the NF2 locus as the germline event.
The mast cell: a neuroimmunoendocrine master player.
Loss of CBP acetyltransferase activity by PHD finger mutations in Rubinstein-Taybi syndrome.
Molecular analysis of the NF2 tumor-suppressor gene in schwannomatosis.
CBP histone acetyltransferase activity regulates embryonic neural differentiation in the normal and Rubinstein-Taybi syndrome brain.
Gross deletions of the neurofibromatosis type 1 (NF1) gene are predominantly of maternal origin and commonly associated with a learning disability, dysmorphic features and developmental delay.
Syndrome of early onset colon cancers, hematologic malignancies & features of neurofibromatosis in HNPCC families with homozygous mismatch repair gene mutations.
Somatic gene mutation and human disease other than cancer.
Defect of histone acetyltransferase activity of the nuclear transcriptional coactivator CBP in Rubinstein-Taybi syndrome.
SARCOMAS OF THE PERIPHERAL NERVES AND SOMATIC SOFT TISSUES ASSOCIATED WITH MULTIPLE NEUROFIBROMATOSIS (VON RECKLINGHAUSEN'S DISEASE).
Histone deacetylase inhibitors: possible implications for neurodegenerative disorders.
Ablation of CBP in forebrain principal neurons causes modest memory and transcriptional defects and a dramatic reduction of histone acetylation but does not affect cell viability.
Nf1 mutation expands an EGFR-dependent peripheral nerve progenitor that confers neurofibroma tumorigenic potential.
Epigenetic dysregulation in cognitive disorders.
CBP, a transcriptional coactivator and acetyltransferase.
Deletion of the entire NF1 gene detected by the FISH: four deletion patients associated with severe manifestations.
Tumors in Rubinstein-Taybi syndrome.
Carotid Stenosis, Dizziness, Raynaud's Disease, Scintillating Scotoma
Rubinstein-Taybi syndrome: clinical and molecular overview.
Malignant transformation and new primary tumours after therapeutic radiation for benign disease: substantial risks in certain tumour prone syndromes.
CLINICAL FEATURES, DIAGNOSIS AND TREATMENT OF PHEOCHROMOCYTOMA: A REVIEW OF 76 CASES.
Genetic heterogeneity in Rubinstein-Taybi syndrome: mutations in both the CBP and EP300 genes cause disease.
Autism and associated medical disorders in a French epidemiological survey.
Truncated CBP protein leads to classical Rubinstein-Taybi syndrome phenotypes in mice: implications for a dominant-negative mechanism.
A mouse model of Rubinstein-Taybi syndrome: defective long-term memory is ameliorated by inhibitors of phosphodiesterase 4.
Chronic myelomonocytic leukemia in childhood: a retrospective analysis of 110 cases. European Working Group on Myelodysplastic Syndromes in Childhood (EWOG-MDS)
p300/CBP and cancer.
Chromatin acetylation, memory, and LTP are impaired in CBP+/- mice: a model for the cognitive deficit in Rubinstein-Taybi syndrome and its amelioration.
Leukemia-associated NF1 inactivation in patients with pediatric T-ALL and AML lacking evidence for neurofibromatosis.
Deregulated Ras signaling in developmental disorders: new tricks for an old dog.
Rubinstein-Taybi syndrome caused by mutations in the transcriptional co-activator CBP.
Ras signaling and therapies.
The IC-SNURF-SNRPN transcript serves as a host for multiple small nucleolar RNA species and as an antisense RNA for UBE3A.
Imprinting in Prader-Willi and Angelman syndromes.
Ube3a is required for experience-dependent maturation of the neocortex.
Mice devoid of gamma-aminobutyrate type A receptor beta3 subunit have epilepsy, cleft palate, and hypersensitive behavior.
The Angelman syndrome candidate gene, UBE3A/E6-AP, is imprinted in brain.
Homologous association of oppositely imprinted chromosomal domains.
The Angelman syndrome ubiquitin ligase localizes to the synapse and nucleus, and maternal deficiency results in abnormal dendritic spine morphology.
Deletions of a differentially methylated CpG island at the SNRPN gene define a putative imprinting control region.
The Angelman syndrome-associated protein, E6-AP, is a coactivator for the nuclear hormone receptor superfamily.
Intracytoplasmic sperm injection may increase the risk of imprinting defects.
Epigenetic overlap in autism-spectrum neurodevelopmental disorders: MECP2 deficiency causes reduced expression of UBE3A and GABRB3.
Dopaminergic system dysregulation in the mrsk2_KO mouse, an animal model of the Coffin-Lowry syndrome.
Coffin-Lowry syndrome: a 20-year follow-up and review of long-term outcomes.
Inherited microdeletions in the Angelman and Prader-Willi syndromes define an imprinting centre on human chromosome 15.
A syndromic form of X-linked mental retardation: the Coffin-Lowry syndrome.
Cognitive function in Coffin-Lowry syndrome.
Imprinted expression of the murine Angelman syndrome gene, Ube3a, in hippocampal and Purkinje neurons.
Dizziness, Raynaud Phenomenon, Scintillating Scotoma, Stroke
Altered neurodevelopment associated with mutations of RSK2: a morphometric MRI study of Coffin-Lowry syndrome.